Blue phases are networks of disclination lines, which occur in cholesteric liquid crystals near the transition to the isotropic phase. They have recently been used for the new generation of fast switching liquid crystal displays. Here we study numerically the steady states and switching hydrodynamics of blue phase I (BPI) and blue phase II (BPII) cells subjected to an electric field.

This work introduces a novel extension of the 0/1 Knapsack Problem in which we consider the existence of so-called forfeit sets. A forfeit set is a subset of items of arbitrary cardinality, such that including a number of its elements that exceeds a predefined allowance threshold implies some penalty costs to be paid in the objective function value. A global upper bound on these allowance violations is also considered.

The aim of this talk is to show how de la Vallee Poussin type interpolation based on Chebyshev zeros of rst kind, can be applied to resize an arbitrary color digital image. In fact, using such kind of approximation, we get an image scaling method running for any desired scaling factor or size, in both downscaling and upscaling. The peculiarities and the performance of such method will be discussed.

Active Brownian particles (ABPs), when subject to purely repulsive interactions, are known to undergo activity-induced phase separation broadly resembling an equilibrium (attraction-induced) gas-liquid coexistence. Here we present an accurate continuum theory for the dynamics of phase-separating ABPs, derived by direct coarse graining, capturing leading-order density gradient terms alongside an effective bulk free energy. Such gradient terms do not obey detailed balance; yet we find coarsening dynamics closely resembling that of equilibrium phase separation.

Background Diagnosis and treatment of 22q11.2 deletion syndrome (22q11.2DS) have led to improved life expectancy and achievement of adulthood. Limited data on long-term outcomes reported an increased risk of premature death for cardiovascular causes, even without congenital heart disease (CHD).

Cell death is as important as cell proliferation for cell turn-over, and susceptibility to cell death is affected by a number of parameters that change with time. A time-dependent derangement of such a crucial process, or even the simple cell loss mediated by cell death impinges upon aging and longevity. In this review we will discuss how cell death phenomena are modulated during aging and what is their possible role in the aging process.